marcks protein plays a crucial role in signal transduction pathways within eukaryotic cells.
marcks蛋白在真核细胞的信号转导通路中发挥关键作用。
the phosphorylation of marcks regulates its interaction with actin filaments and modulates cell motility.
marcks的磷酸化调节其与肌动蛋白丝的相互作用并调节细胞运动性。
researchers have identified marcks as a key substrate for protein kinase c in various cell types.
研究人员已确定marcks是多种细胞类型中蛋白激酶c的关键底物。
marcks expression is significantly upregulated during neuronal development and synaptic plasticity.
marcks表达在神经元发育和突触可塑性过程中显著上调。
mutations in the marcks gene have been associated with neurodevelopmental disorders in clinical studies.
临床研究已将marcks基因突变与神经发育障碍联系起来。
the marcks protein contains multiple its sub phosphorylation sites that controlcellular localization.
marcks蛋白含有多个控制其亚细胞定位的磷酸化位点。
inhibiting marcks function through pharmacological approaches affects cell migration and invasion.
通过药理学方法抑制marcks功能会影响细胞迁移和侵袭。
marcks is targeted to the plasma membrane through n-terminal myristoylation and electrostatic interactions.
marcks通过n端豆蔻酰化和静电相互作用被靶向到质膜。
calcium influx triggers rapid phosphorylation of marcks by downstream kinases.
钙内流会触发下游激酶对marcks的快速磷酸化。
marcks interacts with calmodulin in a calcium-dependent manner, regulating various cellular processes.
marcks以钙依赖性方式与钙调蛋白相互作用，调节各种细胞过程。
the knockdown of marcks using sirna results in dramatic changes to cell morphology and adhesion.
使用sirna敲减marcks会导致细胞形态和粘附发生显著变化。
marcks serves as a molecular hub integrating signals from multiple signaling pathways.
marcks作为分子枢纽整合来自多个信号通路的信号。
marcks protein plays a crucial role in signal transduction pathways within eukaryotic cells.
marcks蛋白在真核细胞的信号转导通路中发挥关键作用。
the phosphorylation of marcks regulates its interaction with actin filaments and modulates cell motility.
marcks的磷酸化调节其与肌动蛋白丝的相互作用并调节细胞运动性。
researchers have identified marcks as a key substrate for protein kinase c in various cell types.
研究人员已确定marcks是多种细胞类型中蛋白激酶c的关键底物。
marcks expression is significantly upregulated during neuronal development and synaptic plasticity.
marcks表达在神经元发育和突触可塑性过程中显著上调。
mutations in the marcks gene have been associated with neurodevelopmental disorders in clinical studies.
临床研究已将marcks基因突变与神经发育障碍联系起来。
the marcks protein contains multiple its sub phosphorylation sites that controlcellular localization.
marcks蛋白含有多个控制其亚细胞定位的磷酸化位点。
inhibiting marcks function through pharmacological approaches affects cell migration and invasion.
通过药理学方法抑制marcks功能会影响细胞迁移和侵袭。
marcks is targeted to the plasma membrane through n-terminal myristoylation and electrostatic interactions.
marcks通过n端豆蔻酰化和静电相互作用被靶向到质膜。
calcium influx triggers rapid phosphorylation of marcks by downstream kinases.
钙内流会触发下游激酶对marcks的快速磷酸化。
marcks interacts with calmodulin in a calcium-dependent manner, regulating various cellular processes.
marcks以钙依赖性方式与钙调蛋白相互作用，调节各种细胞过程。
the knockdown of marcks using sirna results in dramatic changes to cell morphology and adhesion.
使用sirna敲减marcks会导致细胞形态和粘附发生显著变化。
marcks serves as a molecular hub integrating signals from multiple signaling pathways.
marcks作为分子枢纽整合来自多个信号通路的信号。
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