| Plural | hexanucleotides |
hexanucleotide repeat
שכפול הексנוקליאוטיד
hexanucleotide expansion
הרחבה של הексנוקליאוטיד
expanded hexanucleotide
הексנוקליאוטיד המורחב
hexanucleotide sequence
רצף הексנוקליאוטיד
mutant hexanucleotide
הексנוקליאוטיד המוטנטי
hexanucleotide repeats
שכפלים של הексנוקליאוטיד
pathogenic hexanucleotide
הексנוקליאוטיד פתוגני
hexanucleotide tract
tract של הексנוקליאוטיד
repeating hexanucleotide
הексנוקליאוטיד המ повтор
hexanucleotide motif
מוטיו של הексנוקליאוטיד
the hexanucleotide repeat expansion causes a severe neurological disorder.
scientists discovered a hexanucleotide repeat mutation in the c9orf72 gene.
the hexanucleotide repeat sequence is associated with als and frontotemporal dementia.
researchers are investigating hexanucleotide repeat therapeutics.
hexanucleotide expansions can be detected through genetic testing.
the hexanucleotide repeat length varies among patients.
toxic proteins are produced from hexanucleotide repeat rna.
hexanucleotide-mediated toxicity has been observed in cellular models.
the hexanucleotide mutation is the most common genetic cause of als.
antisense oligonucleotides target hexanucleotide repeat transcripts.
hexanucleotide repeats form abnormal rna structures.
the hexanucleotide expansion size correlates with disease onset.
hexanucleotide repeat
שכפול הексנוקליאוטיד
hexanucleotide expansion
הרחבה של הексנוקליאוטיד
expanded hexanucleotide
הексנוקליאוטיד המורחב
hexanucleotide sequence
רצף הексנוקליאוטיד
mutant hexanucleotide
הексנוקליאוטיד המוטנטי
hexanucleotide repeats
שכפלים של הексנוקליאוטיד
pathogenic hexanucleotide
הексנוקליאוטיד פתוגני
hexanucleotide tract
tract של הексנוקליאוטיד
repeating hexanucleotide
הексנוקליאוטיד המ повтор
hexanucleotide motif
מוטיו של הексנוקליאוטיד
the hexanucleotide repeat expansion causes a severe neurological disorder.
scientists discovered a hexanucleotide repeat mutation in the c9orf72 gene.
the hexanucleotide repeat sequence is associated with als and frontotemporal dementia.
researchers are investigating hexanucleotide repeat therapeutics.
hexanucleotide expansions can be detected through genetic testing.
the hexanucleotide repeat length varies among patients.
toxic proteins are produced from hexanucleotide repeat rna.
hexanucleotide-mediated toxicity has been observed in cellular models.
the hexanucleotide mutation is the most common genetic cause of als.
antisense oligonucleotides target hexanucleotide repeat transcripts.
hexanucleotide repeats form abnormal rna structures.
the hexanucleotide expansion size correlates with disease onset.
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